Best Brain Foods: Greens and Beets Put to the Test
Cocoa and nitrite-rich vegetables, such as green leafies and beets, are put to the test for cognitive function.
Cocoa and nitrite-rich vegetables, such as green leafies and beets, are put to the test for cognitive function.
Randomized controlled studies put nuts, berries, and grape juice to the test for cognitive function.
Do the benefits outweigh the risks for acid-blocker drugs (proton pump inhibitors like Nexium/Prilosec/Prevacid)? What about baking soda?
Chicken, fish, and egg powder in processed foods present greater risk from cholesterol oxidation byproducts, but there are things you can do to reduce exposure.
Oxidized cholesterol can be a hundred times more toxic than regular cholesterol, raising additional concerns about foods such as ghee, canned tuna, processed meat, and parmesan cheese.
Why are millions of dollars spent on shark cartilage supplements?
What evidence is there that our meat-sweet diets play a cause-and-effect role in dementia?
The spice saffron is pitted head-to-head against the leading drug for severe Alzheimer’s disease.
What happened when turmeric curcumin was put to the test to see if it could reverse DNA damage caused by arsenic exposure?
Lemon balm may beat out drugs for controlling the symptoms of severe dementia.
Having hypertension in midlife (ages 40 through 60) is associated with elevated risk of cognitive impairment and Alzheimer’s dementia later in life—even more so than having the so-called Alzheimer’s gene.
The link between Parkinson’s and dairy may not be explained just by the pesticides and lactose.
Learn why I recommend 250mg a day of a pollutant-free source of long-chain omega-3 fatty acids.
Neurotoxin contamination of the dairy supply doesn’t explain why the association between Parkinson’s and skim milk consumption is as strong as the disease’s association with whole milk.
In this “best-of” compilation of his last four year-in-review presentations, Dr. Greger explains what we can do about the #1 cause of death and disability: our diet.
Berries counteract the neurotoxic effects of pesticides in vitro, potentially explaining why berry consumption is associated with lower risk of developing Parkinson’s disease.
Why has fish consumption been associated with cognitive impairment and loss of executive function?
If copper is associated with Alzheimer’s disease, what about healthy, whole plant food sources such as nuts, seeds, beans, and whole grains?
Diet may explain the Nigerian Paradox, where they have among the highest rates of the Alzheimer’s susceptibility gene, ApoE4, but among the lowest rates of Alzheimer’s disease.
High-tech advances, such as PET scanning, offer new insight into the role cholesterol plays in both the amyloid cascade and vascular models of the development of Alzheimer’s dementia.
Vegetables such as beets and arugula can improve athletic performance by improving oxygen delivery and utilization. But, what about for those who really need it—such as those with emphysema, high blood pressure, and peripheral artery disease?
Lack of adequate blood flow to the brain due to clogging of cerebral arteries may play a pivotal role in the development and progression of Alzheimer’s dementia.
Neurodegenerative brain changes begin by middle age, underscoring the need for lifelong preventive brain maintenance.
Dr. Greger has scoured the world’s scholarly literature on clinical nutrition and developed this new presentation based on the latest in cutting edge research exploring the role diet may play in preventing, arresting, and even reversing some of our most feared causes of death and disability.
The mercury content in fish may help explain links found between fish intake and mental disorders, depression, and suicide.
One week on a plant-based diet can significantly drop blood levels of homocysteine, a toxin associated with cognitive decline and Alzheimer’s disease. Without vitamin B12 supplementation, though, a long-term plant-based diet could make things worse.
A quick, non-invasive, and inexpensive test for the early detection of Alzheimer’s disease is developed using only a ruler and some peanut butter.
Organic food consumption appears to reduce exposure to pesticide residues and antibiotic-resistant bacteria.
A neurotoxin called BMAA that concentrates in seafood may help explain clusters of Lou Gehrig’s disease.
The neurotoxin BMAA is found in seafood and the brains of Alzheimer’s and ALS victims. Might dietary changes help prevent amyotrophic lateral sclerosis?
Advanced glycation end products in our diet may suppress sirtuin enzyme activity and play a role in age-related brain volume loss.
If foods like berries and dark green leafy vegetables have been found protective against cognitive decline, why aren’t they recognized as such in many guidelines?
The role of the Mediterranean diet in preventing and treating dementia.
Lifestyle changes could potentially prevent hundreds of thousands of cases of Alzheimer’s disease every year in the United States
The tea plant concentrates aluminum from the soil into tea leaves, but phytonutrients in tea bind to the metal and limit its absorption.
How do canned versus germinated beans (such as sprouted lentils) compare when it comes to protecting brain cells and destroying melanoma, kidney, and breast cancer cells.
What a teaspoon a day of the spice turmeric may be able to do for those suffering from Alzheimer’s disease.
Rural India has the lowest validated Alzheimer’s rates in the world. Is it due to the turmeric in their curry, or their largely plant-based diets?
Dr. Greger has scoured the world’s scholarly literature on clinical nutrition and developed this new presentation based on the latest in cutting-edge research exploring the role diet may play in preventing, arresting, and even reversing some of our leading causes of death and disability.
Over-activated TOR signaling may help explain the link between acne and subsequent risk for prostate and breast cancer.