Transcript: Cow’s Milk Casomorphin and Autism
Attacks of apnea, where babies temporarily stop breathing, and muscular atony, where babies go limp, after exposure to cow’s milk may also be explained by extracentral activity of casomorphin, meaning outside the brain. Casomorphin, this opioite-like peptide produced by cow’s milk, is also responsible for triggering pseudo-allergic reactions and other abnormalities seen in crib death. Moreover, similarly to morphine, they delay the gastric emptying time, and so may increase the risk of infants refluxing stomach back up into the lungs.
Thus, it can be said that the so-called milk-apnea effect may consist of several components: an opioid-induced respiration depression, an opioid-induced pseudo-allergic histamine-related respiratory response, an influence on the peripheral nervous system, a cow’s milk-induced reflux, followed by aspiration-induced apnea.
Sudden infant death syndrome is not the only condition linked to these morphine-like compounds. From another medical journal recently, casomorphins liberated from the cow’s milk protein beta-casein are accused of participating in the cause of such conditions as: autism, crib death, type I diabetes, postpartum psychosis, circulatory disorders, and food allergies.
In terms of autism risk; whereas the human casomorphins, found exclusively in the breast milk of women who don’t drink cow’s milk, are associated with normal psychomotor development and muscle tone. In contrast, elevated levels of bovine casomorphin found in cow’s milk based formula-fed infants was associated with a delay in psychomotor development and muscle spasticity. This evidence suggests that the inability of some infants to adequately eliminate bovine casomorphin may be a risk factor for delay in psychomotor development and other diseases such as autism.
To see any graphs, charts, graphics, images, and quotes to which Dr. Greger may be referring watch the above video. This is just an approximation of the audio contributed by veganmontreal.
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