Do the medium-chain triglycerides in coconut oil and the fiber in flaked coconut counteract the negative effects on cholesterol and artery function?
What About Coconuts, Coconut Milk, and Coconut Oil MCTs?
Below is an approximation of this video’s audio content. To see any graphs, charts, graphics, images, and quotes to which Dr. Greger may be referring, watch the above video.
Studies of populations who eat a lot of coconuts are “frequently cited” by those who sell coconut oil “as evidence” that it does not have harmful effects. For example, there was an apparent absence of stroke and heart disease on the island of Kativa. What were they eating? Well, their diets centered around tubers, like sweet potatoes, with fruits, greens, nuts, corn, and beans. Yes, they ate fish a few times a week, but they were eating largely whole food plant-based diets. So, no wonder they may have had such low rates of artery disease, and one of those whole foods was coconut, not coconut oil.
Now, if you go to Pukapuka, they eat even more coconuts. And, there’s even an island where that’s most of what they eat—and they get high cholesterol. What’s a population eating 87% plant-based—red meat, chicken, and eggs only eaten seldomly, no dairy—doing with cholesterol levels over 200? Well, they’re eating all these coconuts every day. What are their disease rates like? We don’t know. There’s no clinical surveys, no epidemiological data, no autopsies. They did do some EKGs, which can sometimes pick up evidence of past heart attacks, and found few abnormalities, but the sample was too small to be a definitive study. And, even if they did have low disease rates, they weren’t eating coconut oil; they were eating coconuts. Coconut oil proponents pointing to these studies is like the high-fructose corn syrup lobby pointing to studies of healthy populations who eat corn on the cob.
Or, the sugar industry pointing to studies on fruit consumption, and saying see, eat all the refined sugar you want. But, fruit has fiber—and so do coconuts. Just like adding psyllium fiber—Metamucil—to coconut oil can help blunt the adverse effects on cholesterol, fiber derived from defatted coconut itself can reduce cholesterol levels as much as oat bran. And, the plant protein in coconut—also missing from the oil—may also help explain why whole coconut may not have the same effects on cholesterol.
Although coconut fat in the form of powdered coconut milk may not have the same effects on cholesterol as coconut oil, frequent consumption—defined as three or more times a week—has been associated with increased risk of vascular disease, stroke, and heart attack. And no wonder, as coconut milk may acutely impair artery function—as badly as a sausage-and-egg McMuffin. They tested three meals, three different meals: a Western high-fat meal, comprised of an egg McMuffin, a sausage McMuffin, and two hash browns, versus a local high-fat meal (this was done in Singapore; so, the more traditional high-fat meal was rice cooked with coconut milk, though there were also anchovies and an egg), vs. the same amount of calories in an unhealthy low-fat meal, comprised of Frosted Flakes, skim milk, and juice.
Here’s the artery function—the ability of the arteries to relax normally—before Mickey D’s, and after, significantly crippling down artery function within hours of consumption, and the same thing with the coconut milk. So, whether mostly meat-and-oil fat, or coconut milk fat, the arteries similarly clamped down, whereas that horrible sugary breakfast had no effect, no bad effect, on artery function, because, as terrible as it was, it had no saturated fat at all—though it also didn’t have any egg, which may have helped.
Coconut oil proponents also try to argue that coconut oil has MCTs—medium chain triglycerides—shorter-chain saturated fats that aren’t as bad as the longer-chain saturated fats in meat and dairy. But you can’t apply the MCT research to coconut oil. Why? MCT oil is composed of MCTs, the medium chain fats, caprylic and capric acid, about 50% of each, whereas those MCTs make up only like 10% of the coconut oil. Most of coconut oil is the cholesterol-raising longer-chain saturated fats, lauric and myristic. “It is therefore inaccurate to consider coconut oil to contain…predominantly [MCTs].” So, you can’t extrapolate from MCT studies to coconut oil.
That’s actually quite a common misconception, that the saturated fat in coconut oil is mainly MCTs. Actually, coconut oil is mainly lauric and myristic, which have potent LDL (bad cholesterol)-raising effects. “Coconut oil should therefore not be advised for people who should or want to reduce their risk of” the #1 killer of U.S. men and women—heart disease.
It’s like how the beef industry loves to argue that beef fat contains stearic acid, a type of saturated fat that doesn’t raise cholesterol. Yeah, but it also has palmitic and myristic that, like lauric, does raise cholesterol. If you compare the effects of different saturated fats, yes, stearic has a neutral effect on LDL, but palmitic, myristic, and lauric shoot it up. And, frankly, so may MCT oil itself, bumping up LDL like 15% compared to control. So, this “[p]opular belief,” spread by the coconut oil industry, that “coconut oil is healthy” is “not supported by [science].”
So, basically “coconut oil should be [treated no] differently than [animal] sources of dietary saturated fat.” The latest review, published in March 2017 in the Journal of the American College of Cardiology, put it even more simply in their recommendations for patients: “Avoid.”
Please consider volunteering to help out on the site.
- Freeman AM, Morris PB, Barnard N, Esselstyn CB, Ros E, Agatston A, Devries S, O'Keefe J, Miller M, Ornish D, Williams K, Kris-Etherton P. Trending Cardiovascular Nutrition Controversies. J Am Coll Cardiol. 2017 Mar 7;69(9):1172-1187.
- Mensink RP, Zock PL, Kester AD, Katan MB. Effects of dietary fatty acids and carbohydrates on the ratio of serum total to HDL cholesterol and on serum lipids and apolipoproteins: a meta-analysis of 60 controlled trials. Am J Clin Nutr. 2003 May;77(5):1146-55.
- Zock PL, Blom WA, Nettleton JA, Hornstra G. Progressing Insights into the Role of Dietary Fats in the Prevention of Cardiovascular Disease. Curr Cardiol Rep. 2016 Nov;18(11):111.
- Eyres L, Eyres MF, Chisholm A, Brown RC. Coconut oil consumption and cardiovascular risk factors in humans. Nutr Rev. 2016 Apr;74(4):267-80.
- Ng CK, Chan AP, Cheng A. Impairment of endothelial function--a possible mechanism for atherosclerosis of a high-fat meal intake. Ann Acad Med Singapore. 2001 Sep;30(5):499-502.
- Cater NB, Heller HJ, Denke MA. Comparison of the effects of medium-chain triacylglycerols, palm oil, and high oleic acid sunflower oil on plasma triacylglycerol fatty acids and lipid and lipoprotein concentrations in humans. Am J Clin Nutr. 1997 Jan;65(1):41-5.
- Lindeberg S, Lundh B. Apparent absence of stroke and ischaemic heart disease in a traditional Melanesian island: a clinical study in Kitava. J Intern Med. 1993 Mar;233(3):269-75.
- Prior IA, Davidson F, Salmond CE, Czochanska Z. Cholesterol, coconuts, and diet on Polynesian atolls: a natural experiment: the Pukapuka and Tokelau island studies. Am J Clin Nutr. 1981 Aug;34(8):1552-61.
- Trinidad TP, Loyola AS, Mallillin AC, Valdez DH, Askali FC, Castillo JC, Resaba RL, Masa DB. The cholesterol-lowering effect of coconut flakes in humans with moderately raised serum cholesterol. J Med Food. 2004 Summer;7(2):136-40.
- Padmakumaran Nair KG, Rajamohan T, Kurup PA. Coconut kernel protein modifies the effect of coconut oil on serum lipids. Plant Foods Hum Nutr. 1999;53(2):133-44.
- Ekanayaka RA, Ekanayaka NK, Perera B, De Silva PG. Impact of a traditional dietary supplement with coconut milk and soya milk on the lipid profile in normal free living subjects. J Nutr Metab. 2013;2013:481068.
- Tuminah S, Sihombing M. Frequent coconut milk intake increases the risk of vascular disease in adults. Universa Medicina Vol 34, No 2 (2015) 15 December 2015.
- Ganji V, Kies CV. Psyllium husk fiber supplementation to the diets rich in soybean or coconut oil: hypocholesterolemic effect in healthy humans. Int J Food Sci Nutr. 1996;47(2):103-10.
Icons created by Marianna Nardella and Creative Stall from The Noun Project.
Motion graphics by Avocado Video.
- animal products
- artery function
- beans
- beef
- cardiovascular disease
- chicken
- cholesterol
- coconut milk
- coconut oil
- coconuts
- corn
- dairy
- eggs
- fast food
- fiber
- fish
- fruit
- greens
- heart disease
- high fructose corn syrup
- industry influence
- LDL cholesterol
- medium-chain fatty acids
- nutrition myths
- nuts
- oils
- palmitic acid
- plant protein
- Plant-Based Diets
- processed foods
- psyllium
- red meat
- rice
- saturated fat
- stearic acid
- stroke
- sugar
- sweet potatoes
- triglycerides
- vegans
- vegetarians
Below is an approximation of this video’s audio content. To see any graphs, charts, graphics, images, and quotes to which Dr. Greger may be referring, watch the above video.
Studies of populations who eat a lot of coconuts are “frequently cited” by those who sell coconut oil “as evidence” that it does not have harmful effects. For example, there was an apparent absence of stroke and heart disease on the island of Kativa. What were they eating? Well, their diets centered around tubers, like sweet potatoes, with fruits, greens, nuts, corn, and beans. Yes, they ate fish a few times a week, but they were eating largely whole food plant-based diets. So, no wonder they may have had such low rates of artery disease, and one of those whole foods was coconut, not coconut oil.
Now, if you go to Pukapuka, they eat even more coconuts. And, there’s even an island where that’s most of what they eat—and they get high cholesterol. What’s a population eating 87% plant-based—red meat, chicken, and eggs only eaten seldomly, no dairy—doing with cholesterol levels over 200? Well, they’re eating all these coconuts every day. What are their disease rates like? We don’t know. There’s no clinical surveys, no epidemiological data, no autopsies. They did do some EKGs, which can sometimes pick up evidence of past heart attacks, and found few abnormalities, but the sample was too small to be a definitive study. And, even if they did have low disease rates, they weren’t eating coconut oil; they were eating coconuts. Coconut oil proponents pointing to these studies is like the high-fructose corn syrup lobby pointing to studies of healthy populations who eat corn on the cob.
Or, the sugar industry pointing to studies on fruit consumption, and saying see, eat all the refined sugar you want. But, fruit has fiber—and so do coconuts. Just like adding psyllium fiber—Metamucil—to coconut oil can help blunt the adverse effects on cholesterol, fiber derived from defatted coconut itself can reduce cholesterol levels as much as oat bran. And, the plant protein in coconut—also missing from the oil—may also help explain why whole coconut may not have the same effects on cholesterol.
Although coconut fat in the form of powdered coconut milk may not have the same effects on cholesterol as coconut oil, frequent consumption—defined as three or more times a week—has been associated with increased risk of vascular disease, stroke, and heart attack. And no wonder, as coconut milk may acutely impair artery function—as badly as a sausage-and-egg McMuffin. They tested three meals, three different meals: a Western high-fat meal, comprised of an egg McMuffin, a sausage McMuffin, and two hash browns, versus a local high-fat meal (this was done in Singapore; so, the more traditional high-fat meal was rice cooked with coconut milk, though there were also anchovies and an egg), vs. the same amount of calories in an unhealthy low-fat meal, comprised of Frosted Flakes, skim milk, and juice.
Here’s the artery function—the ability of the arteries to relax normally—before Mickey D’s, and after, significantly crippling down artery function within hours of consumption, and the same thing with the coconut milk. So, whether mostly meat-and-oil fat, or coconut milk fat, the arteries similarly clamped down, whereas that horrible sugary breakfast had no effect, no bad effect, on artery function, because, as terrible as it was, it had no saturated fat at all—though it also didn’t have any egg, which may have helped.
Coconut oil proponents also try to argue that coconut oil has MCTs—medium chain triglycerides—shorter-chain saturated fats that aren’t as bad as the longer-chain saturated fats in meat and dairy. But you can’t apply the MCT research to coconut oil. Why? MCT oil is composed of MCTs, the medium chain fats, caprylic and capric acid, about 50% of each, whereas those MCTs make up only like 10% of the coconut oil. Most of coconut oil is the cholesterol-raising longer-chain saturated fats, lauric and myristic. “It is therefore inaccurate to consider coconut oil to contain…predominantly [MCTs].” So, you can’t extrapolate from MCT studies to coconut oil.
That’s actually quite a common misconception, that the saturated fat in coconut oil is mainly MCTs. Actually, coconut oil is mainly lauric and myristic, which have potent LDL (bad cholesterol)-raising effects. “Coconut oil should therefore not be advised for people who should or want to reduce their risk of” the #1 killer of U.S. men and women—heart disease.
It’s like how the beef industry loves to argue that beef fat contains stearic acid, a type of saturated fat that doesn’t raise cholesterol. Yeah, but it also has palmitic and myristic that, like lauric, does raise cholesterol. If you compare the effects of different saturated fats, yes, stearic has a neutral effect on LDL, but palmitic, myristic, and lauric shoot it up. And, frankly, so may MCT oil itself, bumping up LDL like 15% compared to control. So, this “[p]opular belief,” spread by the coconut oil industry, that “coconut oil is healthy” is “not supported by [science].”
So, basically “coconut oil should be [treated no] differently than [animal] sources of dietary saturated fat.” The latest review, published in March 2017 in the Journal of the American College of Cardiology, put it even more simply in their recommendations for patients: “Avoid.”
Please consider volunteering to help out on the site.
- Freeman AM, Morris PB, Barnard N, Esselstyn CB, Ros E, Agatston A, Devries S, O'Keefe J, Miller M, Ornish D, Williams K, Kris-Etherton P. Trending Cardiovascular Nutrition Controversies. J Am Coll Cardiol. 2017 Mar 7;69(9):1172-1187.
- Mensink RP, Zock PL, Kester AD, Katan MB. Effects of dietary fatty acids and carbohydrates on the ratio of serum total to HDL cholesterol and on serum lipids and apolipoproteins: a meta-analysis of 60 controlled trials. Am J Clin Nutr. 2003 May;77(5):1146-55.
- Zock PL, Blom WA, Nettleton JA, Hornstra G. Progressing Insights into the Role of Dietary Fats in the Prevention of Cardiovascular Disease. Curr Cardiol Rep. 2016 Nov;18(11):111.
- Eyres L, Eyres MF, Chisholm A, Brown RC. Coconut oil consumption and cardiovascular risk factors in humans. Nutr Rev. 2016 Apr;74(4):267-80.
- Ng CK, Chan AP, Cheng A. Impairment of endothelial function--a possible mechanism for atherosclerosis of a high-fat meal intake. Ann Acad Med Singapore. 2001 Sep;30(5):499-502.
- Cater NB, Heller HJ, Denke MA. Comparison of the effects of medium-chain triacylglycerols, palm oil, and high oleic acid sunflower oil on plasma triacylglycerol fatty acids and lipid and lipoprotein concentrations in humans. Am J Clin Nutr. 1997 Jan;65(1):41-5.
- Lindeberg S, Lundh B. Apparent absence of stroke and ischaemic heart disease in a traditional Melanesian island: a clinical study in Kitava. J Intern Med. 1993 Mar;233(3):269-75.
- Prior IA, Davidson F, Salmond CE, Czochanska Z. Cholesterol, coconuts, and diet on Polynesian atolls: a natural experiment: the Pukapuka and Tokelau island studies. Am J Clin Nutr. 1981 Aug;34(8):1552-61.
- Trinidad TP, Loyola AS, Mallillin AC, Valdez DH, Askali FC, Castillo JC, Resaba RL, Masa DB. The cholesterol-lowering effect of coconut flakes in humans with moderately raised serum cholesterol. J Med Food. 2004 Summer;7(2):136-40.
- Padmakumaran Nair KG, Rajamohan T, Kurup PA. Coconut kernel protein modifies the effect of coconut oil on serum lipids. Plant Foods Hum Nutr. 1999;53(2):133-44.
- Ekanayaka RA, Ekanayaka NK, Perera B, De Silva PG. Impact of a traditional dietary supplement with coconut milk and soya milk on the lipid profile in normal free living subjects. J Nutr Metab. 2013;2013:481068.
- Tuminah S, Sihombing M. Frequent coconut milk intake increases the risk of vascular disease in adults. Universa Medicina Vol 34, No 2 (2015) 15 December 2015.
- Ganji V, Kies CV. Psyllium husk fiber supplementation to the diets rich in soybean or coconut oil: hypocholesterolemic effect in healthy humans. Int J Food Sci Nutr. 1996;47(2):103-10.
Icons created by Marianna Nardella and Creative Stall from The Noun Project.
Motion graphics by Avocado Video.
- animal products
- artery function
- beans
- beef
- cardiovascular disease
- chicken
- cholesterol
- coconut milk
- coconut oil
- coconuts
- corn
- dairy
- eggs
- fast food
- fiber
- fish
- fruit
- greens
- heart disease
- high fructose corn syrup
- industry influence
- LDL cholesterol
- medium-chain fatty acids
- nutrition myths
- nuts
- oils
- palmitic acid
- plant protein
- Plant-Based Diets
- processed foods
- psyllium
- red meat
- rice
- saturated fat
- stearic acid
- stroke
- sugar
- sweet potatoes
- triglycerides
- vegans
- vegetarians
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What About Coconuts, Coconut Milk, and Coconut Oil MCTs?
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Content URLDoctor's Note
Okay, but doesn’t saturated fat boost HDL, the so-called good cholesterol? Check out Coconut Oil and the Boost in HDL “Good” Cholesterol.
Isn’t coconut oil supposed to be good for Alzheimer’s, though? See Does Coconut Oil Cure Alzheimer’s?.
What about swishing it around in your mouth? See:
- Does Oil Pulling Help with Cancer?
- Oil Pulling Benefits for Plaque and Gingivitis
- Oil Pulling for Teeth Whitening and Bad Breath Tested
If you want to learn more about the original McMuffin artery studies, see The Leaky Gut Theory of Why Animal Products Cause Inflammation.
You may also be interested in Coconut Oil and Abdominal Fat.
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